By Icon Health Publications
This can be a 3-in-1 reference booklet. It provides an entire clinical dictionary protecting 1000's of phrases and expressions in terms of acute myeloid leukemia. It additionally offers wide lists of bibliographic citations. eventually, it presents details to clients on the best way to replace their wisdom utilizing a number of net assets. The publication is designed for physicians, clinical scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with study devoted to acute myeloid leukemia. in case your time is effective, this booklet is for you. First, you won't waste time looking out the net whereas lacking loads of suitable details. moment, the e-book additionally saves you time indexing and defining entries. ultimately, you won't waste time and cash printing 1000's of websites.
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Extra resources for Acute Myeloid Leukemia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
This will be accomplished by transfecting rno into HL-60 cells and determining the effect of rno expression on cell growth and differentiation. The subcellular distribution of rno will be determined using a rno Green Fluorescent Protein fusion gene. rno will be expressed in E. coli and rno protein will be purified to study its effects on RNAse function. Antisera to rno will be raised for further functional studies. rno expression will be determined in normal hematopoietic cells at different stages of differentiation.
We will establish myeloid progenitor cell lines with inducible expression of mutant genes, which will enable a comprehensive investigation of the molecular events mediating dysregulatory effects of mutant neutrophil elastase and mutant G-CSFR. These models will be useful for development of novel therapeutic strategies for treatment of severe congenital neutropenia and prevention of its evolution to acute myeloid leukemia.
Primarily, the PI will examine the role of the Abi adaptorss in these processes. The availability of mice with targeted deletions of abi-1 and abi-2 will greatly facilitate the elucidation of the functional roles of these adaptors in normal and transformed cells. Deregulation of the c-Abl kinase downstream of constitutively activated forms of receptor kinases, Src family kinases and by mutated Abi proteins may play a role in the development of multiple human cancers. Results from the aims proposed will shed light on the biological role(s) of c-Abl and allow for a better understanding of the pathophysiological consequences of deregulated Abl tyrosine kinase activity in human cancers.