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By Richard T. Walker (auth.), Erik De Clercq, Richard T. Walker (eds.)

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De Clercq, J. Balzarini, D. Madej, F. J. Robins, J. Med. , 30, 481 (1987). E. J. Robins, Antimicrob. , 30, 719 (1986). I. L. Peale, Biochem. Biophys. Res. , 81, 521 (1978). I. L. , 6, 193 (1979). B. A. A. Fyle, P. de Miranda, L. J. Schaeffer, Proc. Natl. Acad. , USA, 74, 5716 (1977). J. --seauchamp,~ de-Miranda, ~B. J. Bauer and P. Collins, Nature, 272, 583 (1978). J. Schaeffer, Am. J. , 7~#lA), 4 (1982). For reviews see:-(a) A~ Feddian, D. M. A. , 4, 99 (1984); (b) R. Dolin, Science, 227, 1296 (1985); (c) ~Corey and P.

6 fold greater amount of ribavirin 5'-triphosphate than in comparable uninfected cells which had been treated with similar concentrations of ribavirin. Of considerable interest is the recent report(124) of the increased survival time of mice infected with encephalomyocarditis virus when treated with ribavirin. Ribavirin effectively inhibited myocardial virus replication and reduced viral myocardial damage in the hearts of these test animals(124). 2-a-D-Ribofuranosylselenazole-4-carboxamide (Selenazofurin, 22).

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